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Video Endoscopic Sequence 1 of 10.
Extrinsic compression due to malign ascites
This 34 year-old male that, two years previously was diagnosed as having colon cancer, now present a severe abdominal bulking due to a malign ascites.
For more endoscopic details download the video clips by clicking on the endoscopic images, wait to be downloaded complete then press Alt and Enter; thus you can observe the video in full screen.
All endoscopic images shown in this Atlas contain video clips.
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Video Endoscopic Sequence 2 of 10.
At the gastric fundus is observed two extrinsic compression
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Video Endoscopic Sequence 3 of 10.
In order to relief the ascites a transgastric procedure was performed, first a pre-cut needle was used through an duodenoscope.
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Video Endoscopic Sequence 4 of 10.
After the gastric walls was opened a hydrostatic balloon was used to dilate the small hole.
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Video Endoscopic Sequence 5 of 10.
The gastric wall was open using a sphincterotome, the video clip shows the ascites draining across the gastric wall, the Intra-abdominal pressure was overcome.
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Video Endoscopic Sequence 6 of 10.
A pulsatile bleeding emerging from the gastric wall.
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Video Endoscopic Sequence 7 of 10.
To perform the hemostasis the argon plasma coagulator was used combined with the absolute alcohol.
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Video Endoscopic Sequence 8 of 10.
Injection therapy with absolute alcohol.
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Video Endoscopic Sequence 9 of 10.
After the gastric wall is open, a transgastric endoscopic access of the peritoneal cavity is seen in the video clip.
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Video Endoscopic Sequence 10 of 10.
A transgastric periteneoscopy, a part of the peritoneal cavity is observed.
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Video Endoscopic Sequence 1 of 8.
Gastric Carcinoid Tumor.
Carcinoids are the most common neuroendocrine tumors. The tumor is derived from primitive stem cells in the gut wall but can be seen in the liver, pancreas, bronchus, and ovaries. In children, most cases occur in the appendix, and most are benign and asymptomatic.
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Video Endoscopic Sequence 2 of 8.
Gastric Carcinoid Tumor.
These tumors have a yellow, tan, or gray-brown appearance that can be observed through the intact mucosa. The yellow color is a result of cholesterol and lipid accumulation within the tumor. Tumors can have a polypoid appearance and occasionally can ulcerate.
Similar images of Duodenal Carcinoid Tumor are found in duodenal miscellaneous chapter.
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Video Endoscopic Sequence 3 of 8.
Gastric Carcinoid Tumor.
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Video Endoscopic Sequence 4 of 8.
Gastric Carcinoid Tumor.
Indigo Carmin Stain.
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Video Endoscopic Sequence 5 of 8.
Cromogranina.
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Video Endoscopic Sequence 6 of 8.
4x.
Gastric Carcinoid Tumor.
At low power there is an intramucosal neoplasia.
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Video Endoscopic Sequence 7 of 8.
10x.
Carcinoid Tumor.
At medium power the organoid neoplasia replace the gastric glands. carcinoid tumor.
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Video Endoscopic Sequence 8 of 8.
40x.
The appendix is the most common site of gut carcinoid tumor, followed by the small intestine, rectum, stomach and ileum. Carcinoid tumor are potentially malignant and the tendency of malignant behavior correlate with the site of origin, the depth of local penetration and the size of the tumor.
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Video Endoscopic Sequence 1 of 2.
Gastric Angiodysplasia
This 91-year old lady had medical history of has multiple hospitalizations due to upper gastrointestinal bleeding
Angiodysplasia is ectasia of intestinal submucosal veins and overlying mucosal capillaries. Lesions occur most often in the stomach, followed by the duodenum. Most lesions are less than 10mm in size, and multiple lesions are frequent. Concomitant lesions in the colon occur in 1/3 of cases. Anatomic clustering of acquired or sporatic angiodysplasia is a well described observation.
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Video Endoscopic Sequence 2 of 2.
The endoscopic image as well as the video clip show multiple angiodysplasias
Gastric angiodysplasia is associated with several systemic diseases, most notably hereditary hemorrhagic telangiectasia (HHT), or Rendu-Osler-Weber syndrome.
The first approach to therapy should be to replace and maintain iron. Endoscopic control of bleeding lesions (sclerotherapy, contact probes, and lasers) is well described but the recurrent bleeding rate is high.
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Video Endoscopic Sequence 1 of 3.
Gastric Angiodysplastic lesions and argon plasma coagulation treatment.
Gastric Angiodysplastic lesions may be encountered at any location, but they tend to occur primarily within the corpus. The tree images and video display ablative therapy with argon plasma coagulation APC.
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Video Endoscopic Sequence 2 of 3.
Gastric Angiodysplastic lesions with argon plasma coagulation treatment. Argon plasma coagulation (APC) is a method ofcauterizing the vascular abnormality using a non-contact probe.
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Video Endoscopic Sequence 3 of 3.
Gastric Angiodysplastic lesions with argon plasma coagulation treatment. Argon Plasma Coagulation has been used for more than 10 years in open surgery, laparoscopy and Thoracoscopy, especially for hemostasis of large surface bleeding. It conducts monopolar electrosurgical current to tissue via an ionized argon gas stream (argon plasma).
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Video Endoscopic Sequence 1 of 3.
Foreign body of the stomach that resemble a snake. from time to time the endoscopist get some surprise performing a gastrointestinal endoscopy. 30 year-old woman that had been complaining of severe halitosis that is worse in the morning. The endoscopy was performed in 1990 with the old fiber optics endoscopes (Olympus pre-oes 1T). Therefore the video clips are dark.
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Video Endoscopic Sequence 2 of 3.
We observed the shape that resemble a snake’s head.
Most swallowed foreign bodies pass harmlessly through the gastrointestinal (GI) tract. Foreign bodies that damage the GI tract, become lodged, or have associated toxicity must be identified and removed.
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Video Endoscopic Sequence 3 of 3.
A tooth brush that was found in her stomach.
Impacted foreign bodies.
A foreign body lodged in the GI tract may cause local inflammation leading to pain, bleeding, scarring, and obstruction, or it may erode through the GI tract. Migration from the esophagus most often leads to mediastinitis but may involve the lower respiratory tract or aorta and create an aortoenteric fistula. Migration through the lower GI tract may cause peritonitis.
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Video Endoscopic Sequence 1 of 6.
Heterotopic Pancreas with large central hole.
Heterotopic Pancreas, due to the hole, as the diameter is not usual in the heterotopic pancreas, some biopsies were obtain from the submucosa which was demonstrated by histology that is a pancreas tissue.
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Video Endoscopic Sequence 2 of 6.
Heterotopic pancreas is an uncommon but not an exceedingly rare finding. Most of the time, the heterotopic pancreas is usually small (1 cm to 3 cm in size) and located in the antrum. Pancreatic rests are often discovered incidentally.
Despite the observation that most cases of heterotopic pancreas do not cause problems, the condition has been reported to lead to symptoms from inflammation, obstruction, or even malignant transformation. Smaller rests have been removed using endoscopic techniques, primarily to secure a diagnosis, as the smaller tumors are often asymptomatic.
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Video Endoscopic Sequence 3 of 6.
The biopsies were obtain from the submucosa
Although malignant transformation has been reported, this appears to be uncommon enough that recommendations on follow up probably should be based on symptoms.
With the abuse of alcohol has been reported "gastric pancreatitis."
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Video Endoscopic Sequence 4 of 6.
Low power view of gastric heterotopic pancreas
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Video Endoscopic Sequence 5 of 6.
Acinar pancratic tissue at the left
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Video Endoscopic Sequence 6 of 6.
High power detail of the heterotopic pancreas
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Heterotopic Pancreas
The majority of the patients are asymptomatic and the lesion is diagnosed incidentally.
Heterotopic pancreas is defined as pancreatic tissue outside the boundaries of the pancreas that lacks anatomic and vascular continuity to this organ. Heterotopic pancreas is a relatively infrequent lesion.
The pathogenesis of this lesion is unknown; it is believed to arise during embryonic development of the gastrointestinal tract. The normal pancreas is derived from several evaginations originating from the wall of the primitive duodenum. During embryogenesis, if one or more evaginations remain in the wall of the bowel, then it may be carried away from the remainder of the gland by the developing gastrointestinal tract and may give rise to heterotopic pancreas.
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Pancreatic Heterotopia.
Antral nodule with typically central depression and intact overlying, antral mucosa, the submucosa is the most frequent location, both exocrine and endocrine pancreatic tissue may comprise the lesion. The most distinctive heterotopic lesions occurs in the antrum. Ectopic pancreas generally has a typical apical dimple characteristic of this lesion. A rudimentary ductal system may empty into this depression.
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Video Endoscopic Sequence 1 of 7.
Watermelon Stomach
longitudinal erythymatous stripes that formed lines within the antrum radiating towards the pylorus resembling the stripes of a watermelon and hence the name gastric antral vascular ectasias (GAVE), or watermelon stomach.
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Video Endoscopic Sequence 2 of 7.
Treatment of watermelon stomach (GAVE syndrome) with endoscopic argon plasma coagulation (APC).
The diagnosis is based on the endoscopic findings. The typical lesions have longitudinal rugal folds traversing the antrum and converging on the pylorus, each containing a visible convoluted column of vessels, the aggregate resembling the stripes of a watermelon. Although these lesions are confined to the antrum in the majority of cases, up to 33% of the patients have proximal gastric involvement typically in the presence of a diaphragmatic hernia. It is important to emphasize, however, that these lesions might be misdiagnosed as gastritis or portal gastropathy and thus delay in treatment could result.
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Video Endoscopic Sequence 3 of 7.
Watermelon stomach is an increasingly recognizable cause of persistent acute or occult gastrointestinal bleeding, especially in elderly women. The chief presentation is severe iron deficiency anemia and occult or overt gastrointestinal bleeding. Diagnosis is made on endoscopy by the characteristic appearance of visible watermelon linear stripes in the antrum. Histology is rarely needed to confirm the diagnosis. The importance of this lesion lies in the proper recognition since it is amenable to successful therapeutic interventions, leading to endoscopic healing of the lesion, significant improvement in the anemia and a reduction in the need for blood transfusions.
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Video Endoscopic Sequence 4 of 7.
The argon-plasma-coagulation uses instead of laser energy conduction of electric energy by ionized argon gas (plasma), which produces coagulation necrosis of tissues. The potential advantages of the argon-plasma-coagulation lie in the limited deep penetration, which reduces the risk of perforation and the symmetric spread of the coagulation effects in the surrounding mucosa. These properties make the argon plasma-coagulation a promising tool for the endoscopic therapy of mucosal lesions of the GI-tract. Further attractive is the low cost of the argon-plasma -coagulation equipment compared with laser devices.
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Video Endoscopic Sequence 5 of 7.
The therapeutic options are numerous for this condition and needs to be individualized. The simplest form of therapy is iron supplementation and occasional blood transfusions. When these measures fail, other approaches are warranted, including endoscopic, pharmacologic or surgical therapies.
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Video Endoscopic Sequence 6 of 7.
Argon Plasma Coagulator is a new device that allows for non-contact monopolar coagulation of bleeding surfaces, and devitalization of tissue in the gastrointestinal tract. It is safer and much less expensive than lasers, more effective than bipolar cauterization techniques.
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Video Endoscopic Sequence 7 of 7.
The electrode in the argon channel of the probe is connected to an electrosurgical generator.
The APC probe ionizes the argon gas where it remains ionized approximately 2-10mm distal to the tip of the probe. Ionized Argon gas is electrically conductive. This allows the current to flow between the probe and the tissue. Current density upon arrival at the tissue surface causes coagulation. The application of the energy to the tissue is uniform, and contact free. The Argon plasma beam acts not only in a straight line (axially) along the axis of the probe, but also laterally and radially and "around the corner" as it seeks conductive bleeding surfaces. Following physical principles, the plasma beam has a tendency to turn away from already coagulated (high impedance) areas toward bleeding or still inadequately coagulated receiving treatment. This automatically results in evenly applied, uniform surface coagulation.
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Watermelon Stomach
Watermelon-stomach is a rare cause of gastrointestinal bleeding. There has been an increasing number of reports on the association of this lesion with diseases of the scleroderma group.
Gastric antral vascular ectasia (GAVE), also referred to as Watermelon stomach, is a severe haemorrhagic condition that leads to significant morbidity and transfusion dependence in some patients. Re-bleeding following treatment is common, and there are few treatment options. Until recent treatment modalities were developed, the only options available to patients were blood transfusions or the surgical removal of the stomach (antrectomy). The estimated prevalence of GAVE ranges from 0.3 per cent of cases in a large endoscopic series to 4 per cent in highly selected cohorts with severe gastrointestinal bleeding. Although some patients with diffuse GAVE may have portal hypertensive gastropathy, for the purpose of this application the indication is GAVE not related to portal hypertensive gastropathy.
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Video Endoscopic Sequence 1 of 3.
Upside-Down Stomach
(Gastric Rotation)
The term "gastric volvulus" is reserved for cases in which the abnormal rotation has led to strangulation and obstruction Gastric volvulus is defined as an abnormal rotation of the stomach of more than 180°, creating a closed loop obstruction that can result in incarceration and strangulation.
The stomach can rotate along an axis that is 90° to the longitudinal axis. Such rotation is called a mesenteroaxial rotation . This rotation may lead to an upside-down stomach. Mesenteroaxial rotation of an intrathoracic stomach is less common than organoaxial rotation. Mesenteroaxial rotation is more frequently seen in patients with progression of a type 2 paraesophageal hiatal hernia.
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Video Endoscopic Sequence 2 of 3.
The most common cause of gastric volvulus in adults is diaphragmatic defects. In cases of paraesophageal hernias, the gastroesophageal junction remains in the abdomen while the stomach ascends adjacent to the esophagus, resulting in an upside-down stomach. Gastric volvulus is the most common complication of paraesophageal hernias.
It has also been reported to complicate gastroesophageal surgery, neuromuscular disorders, and intra-abdominal tumors. Rarely, gastric volvulus may be a complication of liver transplant and may be related to ligation of the hepatogastric ligament during the hepatectomy.
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Video Endoscopic Sequence 3 of 3.
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Video Endoscopic Sequence 1 of 7.
Non-specific finding in the gastric fundus in a lupus patientīs
This 54 year-old female with systemic lupus erythaematosus presenting with abdominal pain, physical Examination the abdomen soft, nontender, no masses, hernias or organomegaly, two months previously had discontinued her treatment with corticosteroids.
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Video Endoscopic Sequence 2 of 7.
Systemic lupus erythematosus (SLE) is a chronic inflammatory disease of unknown cause that affects multiple organ systems. Immunologic abnormalities, especially the production of a number of antinuclear antibodies, are another prominent feature of this disease. The clinical course is marked by spontaneous remissions and relapses. Its multisystemic manifestations and the complications from the use of immunosuppressive agents make the diagnosis and management of this entity challenging.
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Video Endoscopic Sequence 3 of 7.
Gastric Erosion is observed the biopsies were negative to malignancy.
Gastrointestinal (GI) manifestations are common in patients with systemic lupus erythematosus (SLE). Virtually all patients with SLE require treatment with NSAID therapy and/or corticosteroids
The ulcerogenic effects of NSAIDs and corticosteroids used in combination are synergistic and put the patient at a high risk of serious ulcer disease. In addition, high-dose steroids may mask the early clinical signs of peptic ulcer perforation.
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Video Endoscopic Sequence 4 of 7.
Systemic lupus erythematosus: Gastrointestinal Tract Problems: Impairment of blood supply to various parts of the gastrointestinal tract may result in abdominal pain, damage to the liver or pancreas (pancreatitis), or a blockage or tear (perforation) of the gastrointestinal tract.
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Video Endoscopic Sequence 5 of 7.
The inflammatory infiltrate from patients with SLE was found to contain higher levels of young and mature fibroblasts than those from patients with gastroduodenitis, and was associated with the progression of SLE. During disease exacerbation, immune complex deposition was observed in the arteriolar walls
The inflammatory changes in the gastric and duodenal mucosa were ascertained to be associated with the progression of SLE. In exacerbation of SLE, the walls of vessels (arterioles) exhibited immune complexes classified mainly as IgG and, to a lesser degree, as IgM. In remission, the luminescence of the vessels decreased. The serum level of immunoglobins did not correlate with their regional production in the gastric and duodenal mucosa.
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Video Endoscopic Sequence 6 of 7.
Systemic lupus erythematosus (SLE) on the gastrointestinal (GI) tract from mouth to anus, attempting to distinguish the features that are most likely to be due to therapy. GI manifestations of SLE include mouth ulcers, dysphagia, anorexia, nausea, vomiting, hemorrhage and abdominal pain. GI vasculitis is usually accompanied by evidence of active disease in other organs. Early recognition of the significance of these symptoms offers the best opportunity.
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Video Endoscopic Sequence 7 of 7.
The inflammatory infiltrate from patients with SLE was found to contain higher levels of young and mature fibroblasts than those from patients with gastroduodenitis, and was associated with the progression of SLE. During disease exacerbation, immune complex deposition was observed in the arteriolar walls
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Foreign body.
Chewed gum in the stomach. Patient swallowed the gum while in the waiting room.
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Phytobezoar.
Bezoars are concretions in the GI tract that increase in size by the accumulation of nonabsorbable food or fibers. They are uncommon, but when present, they are usually found in patients with altered GI motility or with a history of gastric surgery. A phytobezoar is composed of indigested plant or vegetable fibres, plant skins and leaves. A phytobezoar may develop when foreign material accumulates in the stomach because of indigestibility, poor mastication or disturbances in the gastric emptying mechanism which can occur following surgical procedures such as vagotomy, pyloroplasty or antrectomy. A trichobezoar is secondary to hair ingestion, usually in mentally disturbed patients
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