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Video Endoscopic Sequence 1 of 10.
Extrinsic compression due to malign ascites
This 34 year-old male that, two years previously was diagnosed as having colon cancer, now present a severe abdominal bulking due to a malign ascites.
For more endoscopic details download the video clips by clicking on the endoscopic images, wait to be downloaded complete then press Alt and Enter; thus you can observe the video in full screen.
All endoscopic images shown in this Atlas contain video clips.
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Video Endoscopic Sequence 2 of 10.
At the gastric fundus is observed two extrinsic compression
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Video Endoscopic Sequence 3 of 10.
In order to relief the ascites a transgastric procedure was performed, first a pre-cut needle was used through an duodenoscope.
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Video Endoscopic Sequence 4 of 10.
After the gastric walls was opened a hydrostatic balloon was used to dilate the small hole.
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Video Endoscopic Sequence 5 of 10.
The gastric wall was open using a sphincterotome, the video clip shows the ascites draining across the gastric wall, the Intra-abdominal pressure was overcome.
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Video Endoscopic Sequence 6 of 10.
A pulsatile bleeding emerging from the gastric wall.
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Video Endoscopic Sequence 7 of 10.
To perform the hemostasis the argon plasma coagulator was used combined with the absolute alcohol.
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Video Endoscopic Sequence 8 of 10.
Injection therapy with absolute alcohol.
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Video Endoscopic Sequence 9 of 10.
After the gastric wall is open, a transgastric endoscopic access of the peritoneal cavity is seen in the video clip.
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Video Endoscopic Sequence 10 of 10.
A transgastric periteneoscopy, a part of the peritoneal cavity is observed.
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Video Endoscopic Sequence 1 of 8.
Gastric Carcinoid Tumor.
Carcinoids are the most common neuroendocrine tumors. The tumor is derived from primitive stem cells in the gut wall but can be seen in the liver, pancreas, bronchus, and ovaries. In children, most cases occur in the appendix, and most are benign and asymptomatic.
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Video Endoscopic Sequence 2 of 8.
Gastric Carcinoid Tumor.
These tumors have a yellow, tan, or gray-brown appearance that can be observed through the intact mucosa. The yellow color is a result of cholesterol and lipid accumulation within the tumor. Tumors can have a polypoid appearance and occasionally can ulcerate.
Similar images of Duodenal Carcinoid Tumor are found in duodenal miscellaneous chapter.
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Video Endoscopic Sequence 3 of 8.
Gastric Carcinoid Tumor.
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Video Endoscopic Sequence 4 of 8.
Gastric Carcinoid Tumor.
Indigo Carmin Stain.
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Video Endoscopic Sequence 5 of 8.
Cromogranina.
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Video Endoscopic Sequence 6 of 8.
4x.
Gastric Carcinoid Tumor.
At low power there is an intramucosal neoplasia.
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Video Endoscopic Sequence 7 of 8.
10x.
Carcinoid Tumor.
At medium power the organoid neoplasia replace the gastric glands. carcinoid tumor.
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Video Endoscopic Sequence 8 of 8.
40x.
The appendix is the most common site of gut carcinoid tumor, followed by the small intestine, rectum, stomach and ileum. Carcinoid tumor are potentially malignant and the tendency of malignant behavior correlate with the site of origin, the depth of local penetration and the size of the tumor.
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Video Endoscopic Sequence 1 of 7.
Severe Bleeding After Biopsies
A 91 year-old male 10 years before underwent a subtotal gastrectomy due to gastric adenocarcinoma of the antrum recently appeared a mass in the pancreato-biliary tree causing jaundice, An endoscopy is performed to looking for tumor regression, near the gastro-jejuno anastomosis had an elevated area that it looks like a scar, multiple biopsies were taken with jumbo forceps causing severe bleeding..
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Video Endoscopic Sequence 2 of 7.
The bleeding was of severe intensity, at the beginning we used argon plasma coagulator.
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Video Endoscopic Sequence 3 of 7.
The argon plasma coagulator was not enough, as it was a large caliber vessel, at this time we made the choice to use between the dual-channel therapeutic endoscope with argon plasma using therapeutic probe (larger caliber) or infiltrate this vessel with absolute alcohol, deciding for the latter.
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Video Endoscopic Sequence 4 of 7.
Injection of absolute alcohol was used in the area of the vessel but also was no successful.
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Video Endoscopic Sequence 5 of 7.
After using the two therapies for hemostatic maneuver, a hemoclip was applied succeeding in stopping the bleeding
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Video Endoscopic Sequence 6 of 7.
Final State of hemostatic therapy
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Video Endoscopic Sequence 7 of 7.
Another image and video clip of Final State of hemostatic therapy.
Important Comments: In the professional practice in almost all therapeutic procedures there are risks of complications as in this case that there was a need of taking biopsies taken as large forceps small malignant lesions often are not demonstrated by the shortage of tissues which is recommended to obtain macro-biopsies but these can take the risk of bleeding in this case, but success of this profession is to manage the complications with certainty, having multiple resources in hand as therapeutic and ablative therapies and homeostatic, the endoscopist should have adequate training and courage as well as the assistants.
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Video Endoscopic Sequence 1 of 7.
Watermelon Stomach
Longitudinal erythymatous stripes that formed lines within the antrum radiating towards the pylorus resembling the stripes of a watermelon and hence the name gastric antral vascular ectasias (GAVE), or watermelon stomach.
Painless occult gastrointestinal bleeding with anemia in an elderly woman is the most typical presentation. This lesion is amenable to endoscopic thermal ablation, and the lesion shown was treated by argon plasma coagulation.
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Video Endoscopic Sequence 2 of 7.
Treatment of watermelon stomach (GAVE syndrome) with endoscopic argon plasma coagulation (APC).
The diagnosis is based on the endoscopic findings. The typical lesions have longitudinal rugal folds traversing the antrum and converging on the pylorus, each containing a visible convoluted column of vessels, the aggregate resembling the stripes of a watermelon. Although these lesions are confined to the antrum in the majority of cases, up to 33% of the patients have proximal gastric involvement typically in the presence of a diaphragmatic hernia. It is important to emphasize, however, that these lesions might be misdiagnosed as gastritis or portal gastropathy and thus delay in treatment could result.
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Video Endoscopic Sequence 3 of 7.
Watermelon stomach is an increasingly recognizable cause of persistent acute or occult gastrointestinal bleeding, especially in elderly women. The chief presentation is severe iron deficiency anemia and occult or overt gastrointestinal bleeding. Diagnosis is made on endoscopy by the characteristic appearance of visible watermelon linear stripes in the antrum. Histology is rarely needed to confirm the diagnosis. The importance of this lesion lies in the proper recognition since it is amenable to successful therapeutic interventions, leading to endoscopic healing of the lesion, significant improvement in the anemia and a reduction in the need for blood transfusions.
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Video Endoscopic Sequence 4 of 7.
The argon-plasma-coagulation uses instead of laser energy conduction of electric energy by ionized argon gas (plasma), which produces coagulation necrosis of tissues. The potential advantages of the argon-plasma-coagulation lie in the limited deep penetration, which reduces the risk of perforation and the symmetric spread of the coagulation effects in the surrounding mucosa. These properties make the argon plasma-coagulation a promising tool for the endoscopic therapy of mucosal lesions of the GI-tract. Further attractive is the low cost of the argon-plasma -coagulation equipment compared with laser devices.
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Video Endoscopic Sequence 5 of 7.
The therapeutic options are numerous for this condition and needs to be individualized. The simplest form of therapy is iron supplementation and occasional blood transfusions. When these measures fail, other approaches are warranted, including endoscopic, pharmacologic or surgical therapies.
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Video Endoscopic Sequence 6 of 7.
Argon Plasma Coagulator is a new device that allows for non-contact monopolar coagulation of bleeding surfaces, and devitalization of tissue in the gastrointestinal tract. It is safer and much less expensive than lasers, more effective than bipolar cauterization techniques.
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Video Endoscopic Sequence 7 of 7.
The electrode in the argon channel of the probe is connected to an electrosurgical generator.
The APC probe ionizes the argon gas where it remains ionized approximately 2-10mm distal to the tip of the probe. Ionized Argon gas is electrically conductive. This allows the current to flow between the probe and the tissue. Current density upon arrival at the tissue surface causes coagulation. The application of the energy to the tissue is uniform, and contact free. The Argon plasma beam acts not only in a straight line (axially) along the axis of the probe, but also laterally and radially and "around the corner" as it seeks conductive bleeding surfaces. Following physical principles, the plasma beam has a tendency to turn away from already coagulated (high impedance) areas toward bleeding or still inadequately coagulated receiving treatment. This automatically results in evenly applied, uniform surface coagulation.
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Watermelon Stomach
Watermelon-stomach is a rare cause of gastrointestinal bleeding. There has been an increasing number of reports on the association of this lesion with diseases of the scleroderma group.
Gastric antral vascular ectasia (GAVE), also referred to as Watermelon stomach, is a severe haemorrhagic condition that leads to significant morbidity and transfusion dependence in some patients. Re-bleeding following treatment is common, and there are few treatment options. Until recent treatment modalities were developed, the only options available to patients were blood transfusions or the surgical removal of the stomach (antrectomy). The estimated prevalence of GAVE ranges from 0.3 per cent of cases in a large endoscopic series to 4 per cent in highly selected cohorts with severe gastrointestinal bleeding. Although some patients with diffuse GAVE may have portal hypertensive gastropathy, for the purpose of this application the indication is GAVE not related to portal hypertensive gastropathy.
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Video Endoscopic Sequence 1 of 3.
Upside-Down Stomach
(Gastric Rotation)
The term "gastric volvulus" is reserved for cases in which the abnormal rotation has led to strangulation and obstruction Gastric volvulus is defined as an abnormal rotation of the stomach of more than 180°, creating a closed loop obstruction that can result in incarceration and strangulation.
The stomach can rotate along an axis that is 90° to the longitudinal axis. Such rotation is called a mesenteroaxial rotation . This rotation may lead to an upside-down stomach. Mesenteroaxial rotation of an intrathoracic stomach is less common than organoaxial rotation. Mesenteroaxial rotation is more frequently seen in patients with progression of a type 2 paraesophageal hiatal hernia.
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Video Endoscopic Sequence 2 of 3.
The most common cause of gastric volvulus in adults is diaphragmatic defects. In cases of paraesophageal hernias, the gastroesophageal junction remains in the abdomen while the stomach ascends adjacent to the esophagus, resulting in an upside-down stomach. Gastric volvulus is the most common complication of paraesophageal hernias.
It has also been reported to complicate gastroesophageal surgery, neuromuscular disorders, and intra-abdominal tumors. Rarely, gastric volvulus may be a complication of liver transplant and may be related to ligation of the hepatogastric ligament during the hepatectomy.
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Video Endoscopic Sequence 3 of 3.
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Video Endoscopic Sequence 1 of 7.
Non-specific finding in the gastric fundus in a lupus patientīs
This 54 year-old female with systemic lupus erythaematosus presenting with abdominal pain, physical Examination the abdomen soft, nontender, no masses, hernias or organomegaly, two months previously had discontinued her treatment with corticosteroids.
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Video Endoscopic Sequence 2 of 7.
Systemic lupus erythematosus (SLE) is a chronic inflammatory disease of unknown cause that affects multiple organ systems. Immunologic abnormalities, especially the production of a number of antinuclear antibodies, are another prominent feature of this disease. The clinical course is marked by spontaneous remissions and relapses. Its multisystemic manifestations and the complications from the use of immunosuppressive agents make the diagnosis and management of this entity challenging.
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Video Endoscopic Sequence 3 of 7.
Gastric Erosion is observed the biopsies were negative to malignancy.
Gastrointestinal (GI) manifestations are common in patients with systemic lupus erythematosus (SLE). Virtually all patients with SLE require treatment with NSAID therapy and/or corticosteroids
The ulcerogenic effects of NSAIDs and corticosteroids used in combination are synergistic and put the patient at a high risk of serious ulcer disease. In addition, high-dose steroids may mask the early clinical signs of peptic ulcer perforation.
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Video Endoscopic Sequence 4 of 7.
Systemic lupus erythematosus: Gastrointestinal Tract Problems: Impairment of blood supply to various parts of the gastrointestinal tract may result in abdominal pain, damage to the liver or pancreas (pancreatitis), or a blockage or tear (perforation) of the gastrointestinal tract.
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Video Endoscopic Sequence 5 of 7.
The inflammatory infiltrate from patients with SLE was found to contain higher levels of young and mature fibroblasts than those from patients with gastroduodenitis, and was associated with the progression of SLE. During disease exacerbation, immune complex deposition was observed in the arteriolar walls
The inflammatory changes in the gastric and duodenal mucosa were ascertained to be associated with the progression of SLE. In exacerbation of SLE, the walls of vessels (arterioles) exhibited immune complexes classified mainly as IgG and, to a lesser degree, as IgM. In remission, the luminescence of the vessels decreased. The serum level of immunoglobins did not correlate with their regional production in the gastric and duodenal mucosa.
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Video Endoscopic Sequence 6 of 7.
Systemic lupus erythematosus (SLE) on the gastrointestinal (GI) tract from mouth to anus, attempting to distinguish the features that are most likely to be due to therapy. GI manifestations of SLE include mouth ulcers, dysphagia, anorexia, nausea, vomiting, hemorrhage and abdominal pain. GI vasculitis is usually accompanied by evidence of active disease in other organs. Early recognition of the significance of these symptoms offers the best opportunity.
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Video Endoscopic Sequence 7 of 7.
The inflammatory infiltrate from patients with SLE was found to contain higher levels of young and mature fibroblasts than those from patients with gastroduodenitis, and was associated with the progression of SLE. During disease exacerbation, immune complex deposition was observed in the arteriolar walls
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Foreign body.
Chewed gum in the stomach. Patient swallowed the gum while in the waiting room.
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Phytobezoar.
Bezoars are concretions in the GI tract that increase in size by the accumulation of nonabsorbable food or fibers. They are uncommon, but when present, they are usually found in patients with altered GI motility or with a history of gastric surgery. A phytobezoar is composed of indigested plant or vegetable fibres, plant skins and leaves. A phytobezoar may develop when foreign material accumulates in the stomach because of indigestibility, poor mastication or disturbances in the gastric emptying mechanism which can occur following surgical procedures such as vagotomy, pyloroplasty or antrectomy. A trichobezoar is secondary to hair ingestion, usually in mentally disturbed patients
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